Download Autoantibodies in Neurological Diseases by G. Martino, A. Vincent (auth.), Angela Vincent, Gianvito PDF

By G. Martino, A. Vincent (auth.), Angela Vincent, Gianvito Martino (eds.)

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Alstead EM, Ritchie JK, Lennard-Jones JE et al (1990) Safety of azathioprine in pregnancy in inflammatory bowel disease. Gastroenterology 99:443-446 47. Buckley LM, Bullaboy CA, Leichtman L, Marquez M (1997) Multiple congenital anomalies associated with weekly low-dose methotrexate treatment of the mother. Arthritis Rheum 40:971-973 48. Fraser FC, Sajoo A (1995) Teratogenic potential of corticosteroids in humans. Teratology 51:45-46 49. Gaudier FL, Santiago-Delin E, Rivera J , Gonzales Z (1988) Pregnancy after renal transplantation.

Osserman KE, Kornfild P, Cohen E et al (1958) Studies in myasthenia gravis. AMA Arch Intern Med 102:72-81 Chapter 3 Immunological Mechanisms in the Lambert-Eaton Myasthenic Syndrome A. A. TONAL! The Lambert-Eaton myasthenic syndrome (LEMS) is an antibody-mediated disorder of neuromuscular and autonomic synaptic transmission. It is paraneoplastic in about 50% of cases, generally being associated with small-cell lung carcinoma (SCLC). In non-cancer cases, it can be associated with other autoimmune diseases [1].

Vincent et al. disease might follow an anti-fetal response. Interestingly, there is growing evidence for the presence of cells of fetal origin in the mother, and it seems likely that these cells, perhaps in the context of a partially foreign major histocompatibility complex (MHC) background, can stimulate the maternal immune system. Nevertheless, the number of women who develop MG during or shortly after their first pregnancy is quite small, and most of the typical early-onset MG patients (onset below 40 years of age) develop MG in their teens.

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